Beta 91a placement test
- 16.12.2021
- Arashigore
- 0.00001441 btc
- 1
The 98AMP is brighter than the 56A. The difference between the 98A and 98AMP is quite big even though they use the same capsule. The 98AMP comes with a built-in head amp and sounds brighter and more present. I have mixed feelings about the Beta My notes say this mic has better low end but the high end is peaky.
Images are big but not sharp. It picks up a lot of room. But later on, when we put this mic on drum overhead, it was a different story. It sounded nice and clean as an overhead mic. It might work on the piano or an acoustic guitar, as well. I guess this one needs more investigation. A microphone that looks this good should sound great. The JZ BT sounded almost too bright. They must be good for overheads. Further investigation recommended. It reacts to well. No wonder this has been a popular choice for both live and recording for decades.
Almost 50 years apart, these two mics sound very similar. In short, no, I was disappointed. It sounded narrow, with peaky high-mids. The sound image was distant and unclear. My notes put the D19 in the bottom. I wanted to approach these recordings like a typical indie session that I or you might do with an artist or band. Stan and I used some standard techniques to close-mic the drums, and we wanted to get sounds quickly, and not fuss too much with mic placement remember time is of the essence.
In addition to hearing the drums on their own, I picked out one of my instrumental songs to play drums to so we could hear how everything sounded in context of a song recording it is about the music right!? After recording with the Beta mics, we later changed the microphones to one my typical recording setups with other mics, to compare sounds with the Betas. Beta 91A The Beta 91A is a boundary microphone that has a solid build and it feels like it could survive a war zone.
It is designed to rest on a pillow or padding inside of the kick drum. It has one option, a contour switch which cuts 7db at Hz. Engaging this switch did add a bit more punch on the top and bottom end, but it was not a dramatic difference. Having the contour switch engaged did sound better however.
We placed the mic about 3 inches from the batter head. We got a great sound quickly that had lots of bottom and a good click sound too. This obviously works on kick drums that have no resonant head, or at least have a hole big enough to get the microphone through.
Kick drums with no hole in the resonant head would require a different microphone. I taped it onto my live room window which essentially turns the entire window pane into a microphone. Here are two examples with the Beta 91A. The first is a mid tempo rock groove played with sticks. The second was a tom driven groove played with blastics. Since the window was at the hight of the cymbals, the cymbals came through a bit strong, but, with some compression and eq, you can give treat these room mics to create some character and depth to a drum sound.
It features an integrated preamplifier, and the A75M microphone mount that clamps onto the rim of the drum. This mic and especially the mount feel solid and well manufactured. The gooseneck of the mic is strong and flexible, and in combination with the mount which features a mic clip on a pivot , allows for quick and easy changes of mic position. This a welcomed change to standard mic clips and mike stands which are often awkward to use when repositioning a microphone.
We put up the Beta 98AMP along side a trusty SM57 on the top side of the snare for comparison, and we were both initially shocked that they sounded quite the same. Upon closer inspection, the Beta 98AMP does have a small high end boost above 8k compared to the SM57, and I found it also has a bit more high hat bleed than the Also, probably due to the clamp mount on the drum, there were low end frequencies even below HZ that translated through the mic.
Putting a 98AMP on the bottom snare head proved also to produce a great sound: plenty of punch and top end too. Oral calcium supplements should be used with caution because of the risk of renal stones. Several bisphosphonates have been used in thalassemia patients for the treatment of osteoporosis with variable results. To date, alendronate, pamidronate, and zoledronate seem to be effective in increasing bone mineral density and normalizing bone turnover, but more controlled trials are necessary to evaluate their efficacy in reducing fracture risks in larger thalassemic populations [ 79 ].
Bone marrow and cord blood transplantation Bone marrow transplantation BMT remains the only definitive cure currently available for patients with thalassemia. The outcome of BMT is related to the pretransplantation clinical conditions, specifically the presence of hepatomegaly, extent of liver fibrosis, history of regular chelation and hence severity of iron accumulation.
The major limitation of allogenic BMT is the lack of an HLA-identical sibling donor for the majority of affected patients. BMT from unrelated donors has been carried out on a limited number of individuals with beta-thalassemia. Provided that selection of the donor is based on stringent criteria of HLA compatibility and that individuals have limited iron overload, results are comparable to those obtained when the donor is a compatible sib [ 81 ]. However, because of the limited number of individuals enrolled, further studies are needed to confirm these preliminary findings.
If BMT is successful, iron overload may be reduced by repeated phlebotomy, thus eliminating the need for iron chelation. Cord blood transplantation from a related donor offers a good probability of a successful cure and is associated with a low risk of GVHD [ 82 , 83 ]. For couples who have already had a child with thalassemia and who undertake prenatal diagnosis in a subsequent pregnancy, prenatal identification of HLA compatibility between the affected child and an unaffected fetus allows collection of placental blood at delivery and the option of cord blood transplantation to cure the affected child [ 84 ].
On the other hand, in cases with an affected fetus and a previous normal child, the couple may decide to continue the pregnancy and pursue BMT later, using the normal child as the donor. Management of thalassemia intermedia Treatment of individuals with thalassemia intermedia is symptomatic [ 4 , 85 ]. As hypersplenism may cause worsening anemia, retarded growth and mechanical disturbance from the large spleen, splenectomy is a relevant aspect of the management of thalassemia intermedia.
Risks associated with splenectomy include an increased susceptibility to infections mainly from encapsulated bacteria Streptococcus Pneumoniae, Haemophilus Influenzae and Neisseria Meningitidis and an increase in thromboembolic events.
Prevention of post-splenectomy sepsis includes immunization against the above mentioned bacteria and antibiotic prophylaxis as well as early antibiotic treatment for fever and malaise. Because of the elevated prevalence of cholelithiasis and the risks of cholecystitis in splenectomised patients, the gallbladder should be inspected during splenectomy and removed in case with or to prevent gallstones.
Treatment of extramedullary erythropoietic masses, detected by magnetic resonance imaging, is based on radiotherapy, transfusions, or hydroxycarbamide. Once leg ulcer has developed, it is very difficult to manage. Regular blood transfusions, zinc supplementation and pentoxifylline, and the use of an oxygen chamber have been proposed for ulcer treatment.
Hydroxycarbamide also has some benefit, either alone or with erythropoietin. Recently promising results have been obtained with platelet derived growth factor. Since patients with thalassemia intermedia have a high risk of thrombosis, exacerbated by splenectomy, it is important to be aware of thrombotic complications.
Supplementary folic acid can be prescribed to patients with thalassemia intermedia to prevent deficiency from hyperactive bone marrow. Lifestyle and diet in beta-thalassemia If the disease is fully compensated by ideal treatment, an individual with thalassemia major can enjoy a near-normal lifestyle and experience normal physical and emotional development from childhood to adulthood, including parenthood. Patients with thalassemia do not have specific dietary requirements, unless they have special prescriptions.
Patients already have a heavy treatment schedule and it is counterproductive to add further restrictions without the likelihood of clear benefit. During growth, a normal energy intake with normal fat and sugar content is recommended. During adolescence and adult life, a diet low in highly refined carbohydrates may be useful in preventing or delaying the onset of impaired glucose tolerance or diabetes.
There is no clear evidence that a specific diet is beneficial in preventing or managing liver disease, unless at late stages. Increased iron absorption from the intestinal tract is characteristic of thalassemia. The amount depends on the degree of erythropoiesis, the Hb level and other potential independent factors. Drinking a glass of black tea with meals reduces iron absorption from food, particularly in thalassemia intermedia [ 85 ]. However, there is no evidence that iron-poor diets are useful in thalassemia major; only foods very rich in iron such as liver, many baby foods, breakfast cereals and multivitamin preparations contain added iron, along with other vitamin supplements should be avoided.
Since many factors in thalassemia promote calcium depletion, a diet containing adequate calcium e. However, nephrolithiasis is seen in some adults with thalassemia major, and calcium supplements should not be given unless there is a clear indication; instead a low oxalate diet should be considered. Patients with thalassemia who remain untransfused or are on low transfusion regimens have increased folate consumption and may develop a relative folate deficiency.
Patients on high transfusion regimens rarely develop this condition, and usually have no need for supplements. Iron overload causes vitamin C to be oxidized at an increased rate, leading to vitamin C deficiency in some patients. Vitamin C may increase iron absorption from the gut, labile iron and hence iron toxicity and may therefore be particularly harmful to patients who are not receiving DFO, as iron mobilized by the vitamin C will remain unbound, causing tissue damage.
The effectiveness and safety of vitamin E supplementation in thalassemia major has not been formally assessed and it is not possible to give recommendations about its use at this time. Patients with thalassemia should be discouraged from consuming alcohol, as it can facilitate the oxidative damage of iron and aggravates the effect of HBV and HCV on liver tissue.
In general, physical activity must always be encouraged unless there is a precise secondary medical condition. Conditions requiring special attention include splenomegaly, severe heart disease and osteoporosis. There is no reason for patients with thalassemia to skip or delay standard recommended vaccinations. To prevent and minimize the risk of infection, immunization with the pneucococcal, Haemophilus Influenza and meningococcal vaccines is recommended about two weeks before splenectomy and after surgery.
It is now universally recognized that thalassemia, like other chronic diseases, has important psychological implications. A key role for treating physicians and other health care professionals is to help patients and families to face up to the difficult demands of treatment, while maintaining a positive role. Therapies under investigation Induction of HbF synthesis can reduce the severity of beta-thalassemia by improving the imbalance between alpha and non-alpha globin chains.
Several compounds including 5-azacytidine, decytabine, and butyrate derivatives gave encouraging results in clinical trials [ 87 ]. These agents induce Hb F by different mechanisms not yet well defined. Their potential in the management of beta-thalassemia syndromes is under investigation. A butyrate derivative, 2,2-Dimethylbutyric acid, sodium salt has received orphan designation for betathalassemia in the United States of America and in Europe. The efficacy of hydroxycarbamide treatment in individuals with thalassemia is still unclear.
Hydroxycarbamide has been used as experimental treatment in patients with thalassemia intermedia to reduce extramedullary masses, to increase Hb levels, and, in some cases, to improve leg ulcers. A good response, correlated with particular polymorphisms in the beta globin cluster i. However, controlled and randomized studies are needed to establish the role of hydroxycarbamide in the management of thalassemia major.
The possibility of correction of the molecular defect in hematopoietic stem cells by transfer of a normal gene via a suitable vector or by homologous recombination is being actively investigated [ 90 ]. The most promising results in the mouse model have been obtained with lentiviral vectors [ 90 , 91 ].
In , orphan designation was granted by the European Commission for autologous haematopoietic stem cells transduced with lentiviral vector encoding the human beta globin gene for the treatment of beta-thalassemia major and intermedia. Prognosis Prognosis of thalassemia minor subjects is excellent. An increased risk for cholelithiasis, especially in association with the Gilbert mutation has been demonstrated [ 92 ].
Patients with thalassemia intermedia who do not usually have severe hemosiderosis are less prone to cardiac problems [ 11 ]. However, pulmonary hypertension, thromboembolic complications, overwhelming postsplenectomy sepsis, and the development of hepatocarcinoma may reduce survival in this group of patients.
The prognosis of betathalassemia major was very grim before there was any treatment available. With no treatment, the natural history was for death by age five from infections and cachexia. The first advance in treatment was the initiation of episodic blood transfusions when the patient was having a particularly bad time. With the advent of this type of therapy, survival was prolonged into the second decade, but it soon became evident that the treatment that saved lives in children caused death from cardiac disease in adolescence or early childhood.
Prognosis for individuals with betathalassemia major has dramatically improved with the advent of DFO. However, many transfusion-dependent patients continued to develop progressive accumulation of iron. This can lead to tissue damage and eventually death, particularly from cardiac disease. Advances in red cell transfusion, and the introduction of new iron chelators and chelation regimes have further prolonged survival in recent years.
Assessment of myocardial siderosis and monitoring of cardiac function combined with intensification of iron chelation have converted a once universally fatal disease to a chronic illness and an excellent long-term prognosis is expected for children who have been chelated since a very young age [ 93 , 94 ]. Bone marrow transplantation is at present the only available definitive cure for patients with thalassemia major.
Conflict of interests The authors declare that they have no competing interests. Authors' contributions RO and RG designed and wrote the paper. Both approved the final version of the manuscript, taking responsibility for the integrity of the data and the accuracy of the data analysis.
Acknowledgements This study was supported by Grants from L. We would like to thank Laura Placido and Daniela Desogus for editorial assistance. The population genetics of the hemoglobinopathies. Bailliere's Clinical Hematology.
Changing patterns of thalassemia worldwide. Ann N Y Acad Sci. Guidelines for the clinical management of thalassemia. Wintrobe's Clinical Hematology. Philadelphia; Thalassemias and related disorders: quantitative disorders of hemoglobin synthesis; pp. Hepatocellular carcinoma in the thalassemia syndromes. Br J Haematol. Survival and complications in thalassemia. Cholelithiasis and Gilbert's syndrome in homozygous beta-thalassemia.
Thalassemia and hypercoagulability. Blood Rev. Final height and endocrine function in Thalassemia Intermedia. J Pediatr Endocrinol Metab. Pregnancy outcome in patients with beta-thalassemia intermedia at two tertiary care centers, in Beirut and Milan. Thalassemia heart disease: a comparative evaluation of thalassemia major and thalassemia intermedia.
Elastic tissue abnormalities resembling pseudoxanthoma elasticum in beta thalassemia and the sickling syndromes. Dominant beta thalassaemia: molecular basis and pathophysiology. Mutations in the general transcription factor TFIIH result in beta- thalassemia in individuals with trichothiodystrophy. Hum Mol Genet. Different substitutions at residue D of the X-linked transcription factor GATA1 lead to altered clinical severity of macrothrombocytopenia and anemia and are associated with variable skewed X inactivation.
HbVar database of human hemoglobin variants and thalassemia mutations: update. Hum Mutat. A Syllabus of Thalassemia Mutations. Galanello R, Cao A. Relationship between genotype and phenotype. Thalassemia intermedia. Genome-wide association study shows BCL11A associated with peristent fetal hemoglobin and amelioration of the phenotype of beta-thalassemia.
Hyperbilirubinaemia in heterozygous b-thalassemia is related to co-inherited Gilbert's syndrome. Cholelithiasis in thalassemia major. Eur J Haematol. Apolipoprotein E epsilon4 allele as a genetic risk factor for left ventricular failure in homozygous beta-thalassemia. Association of heart failure in homozygous beta-thalassemia with the major histocompatibility complex. The influence of hemochromatosis mutations on iron overload of thalassemia major.
Glutathione S-transferase gene polymorphism and cardiac iron overload in thalassemia major. Association of alpha globin gene quadruplication and heterozygous beta thalassemia in patients with thalassemia intermedia.
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| Forex for mac os x | Since many factors in thalassemia promote calcium depletion, a diet containing adequate calcium e. The changes in the distance between the diaphragm and an electrically charged back plate determine the sound. More often than not, I use the 52 for live sound and recording small drums. Maybe it was mic placement, or perhaps it was the mic itself. The frequency levels drop and increase a bit before decaying. The most obvious difference from looking at the wave forms is in the Beta If breakthrough uterine bleeding does not occur, a low oestrogenprogesterone hormone replacement is recommended. |
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By techzelo. It may not be the best single mic for kick drum: but adding it in with another mic like the beta 52 or akg d or audx d6 immediately brings out the snap of the kick. This mic adds tons of definition so you can get a full-bodied kick sound that is more than just boom. Terrific mic. If the drummer doesnt have a porthole in the kick stab one out so you can use this mic.
It's great for quick setup and since I only use the Cajon for the low end as I play it using a foot pedal it works out perfect. I love that I can just throw it in there and it's ready to go. Don't have to worry about a boom stand or anything.
I had the Shure Beta drum mic as well. On a kick drum? Oh yeah. The mic placement starts with a well-tuned drum. This takes into consideration the tuning of the batter head, whether the resonant head has a hole or not, and if there even is a resonant head. If the drum sounds good without a microphone, placing the kick drum mic is a matter of trial and error until you know the drum. Start with the mic placed to once side of the drum, about mid-depth, with the diaphragm facing the beater.
This placement should give you a strong attack while still picking up the low frequencies. The kick drum frequencies on a drum with a resonant head can cancel each other out in undesirable ways. To find the right placement of the mic, move it up or down, forward or back until it sounds good without EQ or compression. Most of the time, I aim the diaphragm toward the inside of the shell to deal with the tubby frequencies that can ruin a good kick drum sound. Condenser Mics vs. Dynamic Condenser microphones use an electric field to pick up the sound.
The changes in the distance between the diaphragm and an electrically charged back plate determine the sound. Dynamic microphones have a magnet and a coil. The coil is connected to the diaphragm, and the differences between the coil and the magnet send the signal to the mixing board or interface. Some microphones need a power supply or batteries. This is almost always the case with condenser mics. Occasionally, you may come across a condenser mic that has its own power supply.
I see this more often in well-established recording studios than in a home recording situation. In the case of the best kick drum mic, I really like my Shure Beta 91a. I use medium duty boom stands for affordability and LP Claws for mic clamps. The Beta 91 sits inside the drum on a hand towel, and the 52 is on a claw. The claw is clamped to a boom stand positioned in front of my kick. It cuts mid range and gives you the high and low end that the kick drum delivers.
Sennheiser makes great microphones. In fact, I will only use the e mics on my toms. They are amazing. The Beta 52a is a supercardioid pattern, which makes it more focused. Audix D6 Another great microphone for kick drum. If I had to own one mic on this list, the Audix D6 is the one because of how well it works with equalization. I want to try a two-mic setup explained at the end of this article and capture the frequencies at their best. The Beta 52 is in the lower right mid-depth of the kick drum with the mic pointing toward the shell of the drum but not directly at it.
This placement worked better to handle some of the cancellation that was happening when I point the mic directly at the beater. Maybe it was mic placement, or perhaps it was the mic itself.
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